Applying cerebral ischemic model induced by 4?vessel occlusion, the contents of mitochondria calaium (Mca), Calmodulin (caM)and the effect of manosidoltetrahexosylganglioside(GM?1) were obseved.

  • 用神经节苷脂(GM1)(10mg/kg)处理器血管闭塞全脑缺血再灌注模型,观察缺血海马组织线粒体钙(MitochondriaCalcium、MCa)钙调素(Calmodulin,CaM)的含量变化及评价GM1对其变化的影响。
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