CONCLUSION: The increase of excitatory amino acids in cortex may be induced by endotoxic shock and may play an important pathophysiological role in the development of shock.

• 结论：内毒素休克可引起脑兴奋性氨基酸含量的持续过度增加，由此而导致的脑功能改变可能在休克发展及转归中起重要的病理生理作用。