Conclusions: The decrease of presynaptic inhibition and formation of new synapses by sprouting of afferent axon terminals probably play a role in spasticity of cerebral palsy.

• 结论：突触前抑制下降、脊神经后根传入纤维侧支出芽、突触再建在脑瘫肌痉挛形成中起了重要的作用；