De Felice et al (20) offer the hypothesis that acute fetal stress and fetal pain can actiate both fetal-placental adrenal and hypothalamic stress hormones leading to premature uterine actiity.

• DeFelice等人提出了一个假说，急性胎儿应激和胎儿疼痛能激活胎儿-胎盘肾上腺素和下丘脑应激激素导致过早的子宫活性。