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- NGF could ease the delayed neuron death (DND) after ischemia reperfusion. NGF可以减轻海马迟发性神经细胞死亡 (DND)性损伤。
- Transient cerebral ischemia may result in degeneration, necrosis, delayed neuron death( DND) and/ or apoptosis. 短暂性脑缺血可出现神经元变性、坏死、迟发性神经元死亡和/凋亡。
- Transient cerebral ischemia may result in degeneration, necrosis, delayed neuron death ( DND) and/or apoptosis. 短暂性脑缺血可出现神经元变性、坏死、迟发性神经元死亡和/或凋亡。
- Transient cerebral ischemia may result in degeneration, necrosis, delayed neuron death( DND) and/ or apoptosis . 短暂性脑缺血可出现神经元变性、坏死、迟发性神经元死亡和/凋亡。
- Ischemia for 6 min resulted in a apparent delayed neuron death (DND) in the hippocampus, while ischemia for 3 min did not cause DND in the hippocampus. 缺血 6min即可导致海马组织明显的神经元延迟性死亡 (delayedneurondeath ,DND) ,而缺血 3min不足以引起海马组织明显的DND。
- Ischemia for 6 min resulted in an apparent delayed neuron death (DND) in the hippocampus, while ischemia for 3 min did not cause DND. 发现缺血 6min即可导致海马组织明显的神经元延迟性死亡 (de layedneurondeath ,DND) ,缺血 3min不足以引起海马组织明显的DND ;
- Since the concept of delayed neuron death (DND)was first brought up at 1982,it is one of the hot spots in which the researchers are interested. 迟发性神经元死亡(DND)自1982年被首次提出以来,始终是研究者关注的热点。
- OGD induced the reduction of GluR2 at postsynaptic membrane and the formation of GluR2 lacking AMPA receptors,which mediated the influx of Ca 2+ and resulted the delayed neuron death. 缺氧损伤可致突触后膜表面GluR2含量降低 ,形成缺乏GluR2的新的AMPA受体通道 ,介导了Ca2 + 的快速内流 ,引起神经元的延迟性死亡
- Advance of Study on Delayed Neuron Death 迟发性神经元死亡的研究进展
- Keywords GluRs;AMPA receptor;delayed neuron death; AMPA受体;神经元延迟性死亡;
- In conclusion,C fos activation after cerebral ischemia may have regulated the delayed neuronal death in the CA 1 region of hippocampus. 脑缺血诱导的c-fos基因表达对于缺血易损海马CA1区迟发性神经元坏死可能起直接的调控作用。
- BackgroundCerebral ischemia reperfusion damage is a kind of delayed neuronal death ( DND). A lot of investigations have showed that DND is closely related to apoptosis. 脑缺血再灌注损伤是一种迟发性神经元死亡(delayed neuronal death,DND),大量研究表明DND与细胞凋亡(cell apoptosis,CA)密切相关。
- Abstract:objective To explore the mechanism leading to delayed neuronal death(DND) and the protective effect of flunarizine on neurons in CA1 area of hippocampus. 摘要:目的 探讨迟发性神经元死亡(DND)发生的机制及氟桂利嗪对海马CA1区神经元的保护作用。
- Purpose: Apoptosis was one of ways that neurons died after Hypoxic-ischemic brain damage (HIBD) . Apoptosis took very important effect on neuronal death in delayed neuronal death (DND). 目的:细胞凋亡是缺氧缺血性脑损伤(Hypoxic-ischemic brain damage,HIBD)过程中神经细胞死亡的一种形式,特别在迟发性神经元死亡(delayed neuronal death,DND)过程中起十分重要的作用。
- Apoptosis is believed to be the main cause of delayed neuronal death (DND) after stroke, and it most likely plays a critical role in the penumbra located in the boundary zone of the cerebral infarts. 近年研究表明脑卒中后迟发性神经元死亡的主要形式是细胞凋亡,且凋亡的细胞主要集中在缺血阴影区(penumbra)。
- Objective: To study the role of nitric oxide (NO) in hippocampal delayed neuronal death (DND) after transient global ischemia and observe the effects of nonselective nitric oxide synthase (NOS) inhibitor N G nitro L arginine(L NNA)on DND. 目的 :研究一氧化氮 ( NO)在缺血性海马迟发性神经元死亡 ( DND)中的作用 ,观察非选择性一氧化氮合酶 ( nitricoxidesynthase,NOS)抑制剂 NG-nitro-L-arginine( L-NNA)对缺血性海马 DND的影响。
- AIM To study the effect of anisodamine on delayed neuronal death (DND) and to investigate the relationship between DND and the changes of ATP, DA(dopamine) contents in striatum during forebrain Ischemia/reperfusion in gerbils. 目的 研究山莨菪碱对沙土鼠脑缺血后海马延迟性神经元死亡的影响并探讨其与纹状体多巴胺和 A T P 含量变化的关系。
- Delayed neuronal death (DND) and selective vulnerability in hippocampus CA1 neurons, which were important characters following ischemia-reperfusion, were also proved to be related with DNA damage and repair. 此外,在脑缺血-再灌注的过程中的重要现象--海马CA1区神经元选择性易损性(Selective vulnerability)和迟发性神经元死亡(Delayed neuronal death,DND)也被证明与DNA损伤及修复有关。
- To observe the effect of platelet activating fortor (PAF) receptor antagonist flolium ginkgo pharmacecs (FGP) on delayed neuronal death (DND) in hippocampus CA1 sector neurons, by using transient brain ischemia mode in gerbils. 实验在沙土鼠短暂性脑缺血模型上应用含有PAF受体拮抗剂的FGP,以观察其对沙土鼠短暂性脑缺血后海马CA1区DND的影响。
- It has still been elusive for effective preventive and therapeutic treatments. Recent years some scholars considered that apoptosis (programmed cell death ,PCD) after SAH may play an important role in the delayed neuronal death(DND). 近年来有学者认为蛛网膜下腔出血(SAH)后凋亡(即程序性细胞死亡,PCD)对迟发性神经元死亡起十分重要的作用。