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- Hippocampus Hypoxia 海马
- Aim: To study the effect of fetus hypobaric hypoxia on the number and channel character of NMDA receptor of hippocampus neurons. 目的:观察低压低氧环境对胎鼠海马神经元NMDA受体数目和通道特性的影响。
- The contents of glycine in whole brain, diencephalon, and hippocampus and brain stem was significantly increased as the animals' tolerance to hypoxia was increased. 结果表明, 随着动物对低氧耐受性的增高, 其全脑, 间脑, 特别是海马,脑干中的甘氨酸含量升高。
- Conclusion: Hippocampus is sensitive to infrasound. 这一效应与次声的声强和暴露时间有关。
- It can prolong mouse s survival time in hypoxia. 醒神液能延长小鼠耐缺氧存活时间。
- Shock, sepsis, acidosis, hypoxia, and so forth, are treated. 对休克，感染，酸中毒，缺氧等应加以治疗。
- A pathological deficiency of oxygen, especially hypoxia. 组织缺氧病理性缺氧，尤指组织缺氧
- Aim:To observe change of binding activity of HIF-1 with erythropoietin(EPO) hypoxia response element(HRE) in the hippocampus of mice preconditioned to hypoxia and explore relationship between the changes and the preconditioning. 目的:观察低氧预适应对小鼠海马组织HIF-1与EPO的低氧应答元件(HRE)结合活性的变化,探讨这种变化与低氧预适应形成的关系。
- Results; NF protein in the hippocampus only labeled some neurons. 结果：在海马结构内并不是所有的神经元均可被神经丝蛋白所标记。
- Results: NF protein in the hippocampus only labeled some neurons. 结果:在海马结构内并不是所有的神经元均可被神经丝蛋白所标记。
- Locating the Impairment of Human Cognitive Function during Hypoxia. 急性缺氧条件下脑认知功能发生障碍的时段。
- Objective: To study serotonin(5-HT) in the hippocampus on sleep. 目的:研究海马中5-羟色胺(5-HT)对大鼠慢波睡眠的影响。
- Hypoxia was the main causeof swelling of mltochondria. 缺氧可导致肝线粒体肿胀。
- The cells have reduced in size or been lost from hypoxia. 由于缺氧细胞已萎缩或消失。
- In contrast, the ECE-2 mRNA level was not affected by hypoxia. 缺氧前后ECE - 2的mRNA水平无明显差异。
- Mothed: the hypoxia bearing was observed in experimental mouse. 方法:对实验小鼠进行耐缺氧等实验观察。
- Objective: To predict the fetus intrauterine hypoxia. 摘要目的：预测胎儿宫内缺氧。
- The experiment group was established by normobaric hypoxia model. 将实验组制成常压缺氧模型。
- Methods: Mice were divided into 3 groups randomly and hippocampus tissue was taken after they were respectively exposed to hypoxia for 4 runs （H4 group），1 run （H1 group） and 0 run （the control group）. 脑虽然对缺氧/缺血极为敏感，但神经细胞对缺氧/缺血有一定的耐受性，即经预先亚致死性的缺氧/缺血暴露，可以增强对随后致死性缺氧/缺血的耐受力，被认为是细胞内源性防护机制的启动[1]，其具体机制尚不十分清楚，但作用效果却很肯定。
- Objective: To investigate toxicity of ethanol extracts from hippocampus. 目的研究海马乙醇提取物的毒性作用。
