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- 这是Shepen,Mut的木乃伊,她曾一度在梯比斯寺院当过歌手。The mummy is that of Shepenmut who was once a singer in the Temple of Thebes.
- 甲基丙二酸血症患儿mut基因两个新遗传突变的发现和鉴证Molecular analysis of two novel mut gene mutations in chinese patient with methylmalonic academia
- 这是Shepen,Mut的木乃伊，她曾一度在梯比斯寺院当过歌手。The mummy is that of Shepenmut who was once a singer in the Temple of Thebes.
- Mut-HPI与改造前的Met-HPI相比，不易被胰酶和羧肽酶B(CPB)降解。Mut-HPI was less easily digested by trypsin and CPB than Met-HPI.
- 实验结果表明,诱导表达48h后,Mut+和MutS重组子表达的产物在SDS-PAGE胶上都出现了清晰的目的带。SDS-PAGE results indicated that there was a clear target band in Muts and Mut+ recombinant Culture supernatant after 48 hours culture respectively.
- 与Mut~+型相比，Mut~s转化子在含营养和色素较少的BMM培养基中即有高表达，且表达产物杂蛋白少，易于纯化。Compared with the Mut~+, the Mut~s-hFL transformants showed higher expression level with less coloring contamination in the product and easier purification for hFL.
- 将此重组表达载体线性化后通过电转化的方法转化PMAD11酵母感受态细胞，筛选Ade~+(Mut~s)重组子并诱导表达。After the recombinant plasmid pMET-PLF-N was linearized and transformed into PMAD11 compentent cells by electroporation, Ade+(Muts)recombinants were selected and induced with methanol.
- 结果表明，诱导培养48小时后，Mut~+重组菌株表达产物在SDS-PAGE胶上显现出清晰的目的蛋白带，而Mut~s重组菌株培养72小时才能显示微弱的目的带；SDS-PAGE results showed that there was a clear target protein band in Mut+ recombinant supernatant after 48 hours of culturing, while a faint band only in Muts recombinant after 72 hours.
- 在含丙烯醇的YPD筛选培养基上筛选获得两株ADH活力降低的突变株mut-1和mut-2,检测突变株mut-1和mut-2的最大ADH活力分别为35.67和43.09U/mL,是原始菌株的41.63%和50.29%。Two mutants,named mut-1 and mut-2,with decreased ADH activity were screened out by yeast peptone dextrose(YPD)agar medium containing allyl alcohol. These two mutants had decreased ADH activities of 41.63%25 and 50.29%25 compared with the parent strain.
- 以同样方法获得的Met-人胰岛素原(Met-HPI)为对照，研究了其理化性质和生物活性的变化，结果表明，Mut-HPI保留了大部分放射免疫活性，而受体结合活性却只有Met-HPI的5.2%。It showed 78.08%25 radio immuno activity (RIA), but only about 5.2%25 of receptor binding activity (RBA) as compared with Met-human proinsulin (Met-HPI) which was prepared in the same way.
- 小干扰RNA对白血病MDR细胞系K562/A02 mdr-1转录、P-gp表达、P-gp功能及K562/A02细胞耐药表型的影响，并进行RNAi的特异性分析。 设计与si-MDR1仅有一个碱基突变的序列（si-MDR1-mut）为对照。An increase of intracellular DNR retention in si-MDR1-treated cells was observed, confirming the inhibition of P-gp function by siRNA targeted MDR1. One base pare mutated control (si-MDR1-Mut) lost the effect of si-MDR1 on both the degradation of mdr1 mRNA and the reduction of P-gp expression.
