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Serum of hepatic injury 肝损伤血清
Methods: Mouse models of hepatic injury were induced by CCl4 and BCG/LPS. Serum levels of ALT and AST, MDA content and the pathological changes of hepatic tissue were observed. 方法采用CCl4及BCG/LPS造成小鼠肝损伤,并测定小鼠血清丙氨酸氨基转移酶(ALT)、天门冬氨酸氨基转移酶(AST)和肝组织丙二醛(MDA)的含量,同时观察肝组织病理改变。
Method: The model of hepatic injury was prepared with distillate spirit administration by gavage, to observe the change of ALT, AST and TG in serum and the GSH, MDA in homogenate. 方法：以白酒灌胃法制作小鼠酒精性肝损伤模型，观察血清丙氨酸氨基转移酶(ALT)、天冬氨酸转氨酶(AST)、三酰甘油(TG)和肝匀浆中还原型谷胱甘肽(GSH)、丙二醛(MDA)等指标的变化。
Hepatic injury mouse model was set up by alloxan.The levels of ALT, AST and AKP in serum were measured and compared, and the pathological changes of hepatic tissues were observed under the microscope. 采用四氧嘧啶致急性肝损伤模型，检测各组小鼠血清中谷转氨酶(ALT)、谷草转氨酶(AST)、碱性磷酸酶(AKP)活性，并对各组小鼠肝脏进行组织病理学观察。
Objective To study the therapeutic results of hepatic injury operations and nonoperative treatmetnts. 目的研究肝脏损伤手术和非手术治疗的治疗效果。
We aimed to assess the clinical, histological and immune features and outcome of hepatic injury in patients with primary hypogammaglobulinemia. 我们旨在评估患者临床以及肝脏损伤病理免疫特征。
METHODS Using CCl 4 to make the model of hepatic injury, and colorimetry to measure the liver function (liver/body ratio,CH,GPT,TG and MDA) of the experimental mice. 方法 :以CCL4造小鼠肝损伤模型 ,用比色法检测两者对肝体比、总胆固醇 (CH)、谷丙转氨酶 (GPT)、甘油三酯 (TG)和丙二醛 (MDA)等指标的抑制作用。
METHODS: Biochemical indexes and histopathological examination of hepatic injury in rats caused by toxicant chemicals D?galactosamine (D?Galn) and CCl?4] were determined. 方法：采用化学毒物D?氨基半乳糖和四氯化碳致大鼠肝脏损伤，测定生化指标及观察病理组织学改变。
The main organ to be involved in the acute poisoning by acetaminophen is the hepar. But it is unknown that the mechanism of hepatic injury induced by acetaminophen . 急性醋氨酚中毒主要受累的器官是肝脏,其引起肝损伤的机制尚不明确。
Conclusion Oncosis is the main pattern of hepatic cells death during I/R injury in cirrhotic rat, and the severity of hepatic injury correlates with the oncosis. 结论胀亡是肝硬化大鼠肝脏I/R损伤肝细胞死亡的主要方式，肝功能损伤与胀亡密切相关。
From four aspects of hepatic injury and NASH pathogenesis, to summarize the change of serology markers of NASH, the purpose is to search the specific diagnosis markers for NASH. 此文从与肝损伤及NASH发病机制相关的四个方面,总结近年文献报道的NASH病变过程中血清学指标的改变,以期找到规律性的变化,为临床血清学诊断NASH提供参考。
Methods Using models of hepatic injury induced by hepatoxicity CCl 4,D-GalN and AAP,together with the technique of TBA,DTNB,CHOD-PAP and IFCC,MDA,GSH-PX,TG and ALP were examined respectively. 方法采用四氯化碳 (CCl4)、半乳糖胺 (D GalN)和对乙酰氨基酚 (AAP)所致小鼠化学性肝损伤模型进行研究 ,分别采用TBA法、DTNB法、CHOD PAP法和IFCC法测定丙二醛 (MDA)、谷胱甘肽 (GSH PX)、甘油三酯 (TG)和碱性磷酸酶 (ALP)。
And in serum of ALT activity was not affected. 而血清中ALT活性无明显影响。
Materials and Methods: 1. We collected the serum of 54 patients withdifferent kinds of hepatitis and 25 patients with HCC and 10 healthy controls. The concentration of IGFBP-3 was measured by ELISA using ACTIVE? IGFBP-3 ELISA kits (DSL-10-6600). 材料与方法:1.;收集不同类型病毒性肝炎、肝硬化以及肝癌患者的血清;用酶联免疫的方法测定血清中IGFBP-3的浓度。
Right branch of hepatic portal v. 肝门静脉右支。
In 1965, hepatitis B virus surface antigen(HBsAg )was founded in the serum of Australian indigene by Blumberg, this was regarded as the start of molecular biological study of hepatitis B virus(HBV). 乙型肝炎病毒(hepatitis B virus ,HBV)分子生物学的研究最早要追溯到1965年，Blumberg等最先在澳大利亚土著人血清中发现了乙型肝炎病毒表面抗原（hepatitis B virus surface antigen,HBsAg ），之后，HBV的基因组结构、基因变异、流行病学特征、致病机理、基因治疗和疫苗等研究进展非常迅速,获得许多重要成果。
Acute hepatic injury induced by thioacetamide. 用硫代乙酰胺复制急性肝损伤模型。
Quantitative study of hepatitis C virus RNA in sera of patien ts with posttransfusion hepatitis C virus infection. 关键词：丙型肝炎病毒；供血者；丙型肝炎病毒定量；丙型肝炎病毒抗体
In order to find out TTV infection in different types of hepatitis, the TTVDNA in serums of 97 cases was detected by nests PCR. 目的:探讨本地区不同型别肝炎患者血清中TTV感染状况。方法:采用巢式PCR对97例不同型别肝炎患者血清中TTVDNA进行检测。
Conclusions:The serum Hyp levels may be an good indicator of hepatic fibrosis. 05),且与肝纤维化程度正相关。