The evidence that glucagon excess contributes to the development and maintenance of fasting hyperglycemia and that failure to suppress glucagon secretion contributes to postprandial hyperglycemia is then reviewed.

• 接下来对证明了胰高糖素的过剩导致餐后高血糖的发生和持续而抑制胰高糖素分泌的功能障碍导致餐后高血糖的发生的相关研究进行了回顾。