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- Methods Cytotoxicity of Ara C was detected by the MTT assay. 方法采用MTT法检测Ara－C的细胞毒作用。
- The tolerance of bone marrow to Ara C was remarkably increased after transfected by drug resistance gene. 耐药基因转染后小鼠骨髓对阿糖胞苷的耐受明显增加。
- CD gene can not only integrate and co express in mice bone marrow cells,but also increase the drug resistance to Ara C. CD基因可以进入小鼠骨髓细胞并且获得共表达,提高了造血细胞对阿糖胞苷的耐药性。
- Objective To investigate the mechanism of HHT and low dose arabinsyl cytosiwe (Ara c) inducing apoptosis of K562 cells. 目的 研究高三尖杉酯碱 (homoharringtonine,HHT)以及小剂量阿糖胞苷 (arabinsylcytosine,Ara C)联合诱导慢粒急变细胞株K5 6 2凋亡作用及其机制。
- Objective:To investigate the effect of ATRA on the apoptosis in HL 60 cells induced by Ara C. 目的 :探讨 HL- 6 0细胞经全反式维甲酸 (ATRA)作用后对化疗药物阿糖胞苷 (Ara- C)诱导凋亡敏感性的变化。
- ADR, VP16, CPT, MTX, Ara C could markedly induce apoptosis of Nalm 6 cells in S phase. ADR、VP16、CPT、MTX和Ara－C能明显诱导细胞凋亡，主要作用于S期。
- Low dose Ara C or harringtonine were given in 16 elderly patients and the CR rate was 22.2%(4/18). 小剂量阿糖胞苷 (Ara C)或三尖杉脂碱 (H)化疗 18例 ;CR率 2 2 .;2%25 (4 18)。
- This study was conducted to demonstrate the effect of IFN, HHT and Ara c eliminating CML Ph +cells. 为观察干扰素 (IFN)、高三尖杉酯碱 (HHT)及阿糖胞苷 (Ara- c)体外对慢性粒细胞性白血病(CML) Ph+ 细胞的生长影响 ,探讨其临床治疗 CML 的有效组合方案。
- Methods:Induction regiment of acute myeloid leukemia (AML) consisted of mitoxantrone and cytosine arabinoside (Ara C). 方法 :急性髓细胞白血病 ( AML)诱导治疗采用 MTZ和阿糖胞苷 ( Ara-C)。
- The protein expressed by Bcl 2 in IL 3+G CSF group is higher than that in G CSF group, G CSF group is lower than Ara c group. Bcl?2表达蛋白IL?3＋G?CSF组高于G?CSF组，G?CSF组高于Ara?c组。
- The combination regimen of HHT and Ara c is effective and economical in patient with late chronic-phase chronic myelogenous leukemia(CML). 高三尖杉酯碱联合低剂量阿糖胞苷治疗慢粒白血病急变简便 ,经济 ,有效
- The conditioning regimens included MACC protocol (Melphalan, Ara C, CTX, and CCNU) in 26 patients and TBI+CY protocol in 5 patients. 移植预处理方案 :2 7例接受MACC(马法兰、阿糖胞苷、环磷酰胺、环己亚硝脲 )方案 ,5例用TBI +CY(全身放疗 +环磷酰胺 )方案 ;
- Liquid cell culture and conventional chromosome assay were used to evaluate the cytotoxic effect of IFN,HHT and Ara c on CML Ph + cell. 以药物作用 2 4小时液体培养及常规染色体分析法研究 IFN、HHT及 Ara- c对 10例 CML 慢性期患者的骨髓中 Ph+ 细胞的生长影响。
- Conclusion HBO enhances the sensitivity of AML cells to Ara c in vitro and increases cell apoptosis of Ara c induced AML. 结论 HBO增强了AML细胞对阿糖胞苷的敏感性 ,使得阿糖胞苷体外诱导AML细胞凋亡增加。
- Objective To investigate apoptosis induced by cytosine arabinoside(Ara C) or teniposide (VM 26 )in fresh leukemic cells from patients with acute non lymphocytic leukemia(ANNL). 目的 探讨Ara－C及VM26治疗白血病的机制。
- Conditioning regimens for second allo HSCT were combination chemotherapy based on moderate dose Ara C (n=5), Bu (n=3), conventional dose Ara C (n=1) and Flud/Mel (n=1). 第二次HSCT时预处理方案包括 :以中剂量阿糖胞苷 (Ara C)为主的联合化疗 5例 ; 以白消安为主的联合化疗 3例 ;
- DA? EA? COMP and HOAP regimens and small dose harringtonine or small dose Ara C were given to the patients according to their disease type and their relevant physical condition. 治疗以小到中剂量 HA、DA、EA、COMP及 HOAP等联合化疗或小剂量三尖杉酯碱、小剂量阿糖胞苷单药化疗。
- Short course high dose mitoxantrone with Ara C demonstrated a higher CR,lower mortality rate and can be applied in the treatment of refractory and relapsed acute leukemia. 短程大剂量米托蒽醌联合阿糖胞苷对难治复发急性白血病有较高的疗效 ,可提高缓解率 ,降低死亡率。
- To observe the effect of Acla and Ara c(AA plan)on acute refractory and recurrent leukemia. 20 patients with acute refractory and recurrent leukemia were treated with AA plan. 观察阿克拉霉素 (Acla)与阿糖胞苷 (Ara- c)联合方案 (AA方案 )治疗难治及复发性急性白血病的疗效 ,对 2 2例难治及复发性急性白血病患者进行了 AA方案化疗。
- It suggests that rhG CSF may enhance the sensitivity of drug resitant AML cells to Ara C in vitro by recruiting blasts into the cell cycle. There for , its use in treating AML Patients of drug resistance with rhG CSF and Ara C was recommended. rhG－CSF可能通过促进耐药AML细胞进入增殖周期，而增强Ara－C对耐药AML细胞的毒性，从而具有耐药的逆转效应，预示着rhG－CSF与Ara－C联合运用治疗耐药AML的潜在价值
