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- Elucidation of the action mechanism and effect of chemokines may find new targets for the prevention and treatment of cerebral ischemic damage. 阐明趋化因子的作用机制和影响,有可能为防治缺血性脑损伤找到新的靶点。
- CONCLUSION Borneol may have a beneficial effect for the treatment of cerebral ischemic damage and its mechanism may be related to improvement of the energy metabolism. 结论冰片注射液对小鼠实验性脑缺血具有保护作用,其机制可能与改善缺血脑组织内能量代谢有关。
- cerebral ischemia damage 脑缺血
- At the same time, cerebral ischemia can activate a complex network of DNA repair tocounteract neuronal DNA damage. 然而缺血在导致DNA损伤的同时,还激活自身的DNA修复系统。
- Hypothermia significantly reduced DND after cerebral ischemia. 低温明显减少脑缺血后的DND。
- Aleppo avens have protective effect to mouse cerebral ischemia. 蓝布不对小鼠脑缺血具有保护作用。
- Bispectral Analysis of EEG Signal during Focal Cerebral Ischemia. 局灶性脑缺血脑电信号的双谱分析
- AIM: To study the effects of clomipramine on cerebral ischemia. 目的:研究氯丙米嗪对脑缺血的作用。
- Effect of Natural Medicines on Cascade Reaction and Reperfusion in Cerebral Ischemic Damage 天然药物对脑缺血损伤级联反应和再灌注的作用
- Method: Rat model of acute local cerebral ischemia was made by ligating middle cerebral artery.The cerebral damage was evaluated by staining with hematoxylin and eosin((HE). 方法:采用闭夹大脑中动脉造成局部脑缺血模型,对实验标本作HE染色后观察其脑组织的病理学改变。
- Objective To discuss the infuence on digestive organs with cerebral ischemia To give the theory ligation support to perserve other organ damage in cerebral ischenia patients. 目的探讨脑缺血对消化器官的影响,为临床脑血管病患者防止其他内脏器官病变提供理论依据。
- EFFECTS OF SELECTIVE BRAIN HYPOTHERMIA ON CEREBRAL ISCHEMIA IN DOGS. 选择性低温对犬脑缺血性损害的保护作用初步探讨。
- Aim To observe the actions of mailuoning oral liquid against cerebral ischemia. 目的观察脉络宁口服液的抗脑缺血作用。
- The mouse acute incomplete cerebral ischemia was observed by ligation method. 小鼠急性不完全性脑缺血采用结扎法;
- Conclusion: TXLC is an effective drug for UAP treatment, which could efficiently regulate the ET and CGRP metabolism and ameliorate the degree of myocardial ischemia damage. 结论:通心络胶囊能有效调节ET、CGRP代谢失衡状况,减少心肌缺血损伤程度,是治疗UAP的有效药物。
- Mongolian gerbil was used as the delayed neuronal damage (DND)animal model, and was submitted to 5-minute cerebral ischemia by bilateral carotid artery occlusion, followed by 1 to 96 hours reperfusion. 本文以结扎沙土鼠双侧项总动脉5min,然后使血流再通的方法制作迟发性神经元损伤动物模型。
- Conclusion HSYA was showed to inhibit the ET release, increase the MBF, improve the cardiac oxygen metabolism, and then inhibit the myocardial ischemia damage. 结论HSYA可以抑制ET的释放,增加心肌血流量,改善心肌氧代谢,抑制心肌缺血损伤。
- Objective: To investigate effects of total paeony glycoside (TPG) on cerebral ischemia mice. 目的:观察赤芍总甙对小鼠实验性脑缺血的影响。
- Objective To study the expression change of GFAP after hypoxia preconditioning and focal cerebral ischemia. 目的探讨缺氧预处理及大鼠局灶性脑缺血后GFAP表达变化。
- Transient cerebral ischemia may result in degeneration, necrosis, delayed neuron death( DND) and/ or apoptosis. 短暂性脑缺血可出现神经元变性、坏死、迟发性神经元死亡和/凋亡。