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effective renal blood flow, ERBF 肾有效血流量
effective renal blood flow 有效肾血流量
ERBF effective renal blood flow 有效肾血流量
The age-related reduction in renal blood flow is accompanied by a gradual loss of functioning glomeruli. 年龄相关性的肾血流量减少并伴随着功能性肾单位的逐渐减少。
The mechanisms of verapamil protection against ARF are: verapamil increased renal blood flow and GFR, improved renal cortical blood circulation, etc. 其机理与增加肾血流量、善肾皮质缺血区域的血液循环等因素有关。
In fact cardiac output or renal blood flow may be above normal at the same time that capillary perfusion is very deficient. 事实上，心输出量，或肾血流量可能就在毛细血管灌注十分不定时，反而高于正常的流量。
There are inappropriate changes of renal hemodynamics that renal blood flow reduce 50%,and glomerular function reduce rapid at the process of ARF. ARF发生时，肾脏存在异常的肾血流动力学的改变，肾血流量减少50%25，肾小球有效滤过压下降。
Abstract： Objective:To study the renal blood flow perfusion and its clinical significance in hydronephrosis by Color Doppler Energy(CDE). 摘要：目的：利用彩色多普勒能量显像技术(CDE)探讨不同程度肾积水患者肾脏的血流灌注及其临床意义。
He found that dopamine increased renal blood flow but did not improe medullary PO2.This finding is most likely due to the inhibition of tubulo-glomerular feedback (TGF). 结果发现，多巴胺增加肾血流但并不增加髓质的PO2，这很可能是因为球管反馈（TGF）抑制的原因。
He found that dopamine increased renal blood flow but did not improve medullary PO2.This finding is most likely due to the inhibition of tubulo-glomerular feedback (TGF). 结果发现，多巴胺增加肾血流但并不增加髓质的PO2，这很可能是因为球管反馈（TGF）抑制的原因。
The earliest abnormal of renal blood flow occured in hypertensives included that the end diastolic velocity (V d) of interlobar artery was decreased and the acceleration time (AT) of segmental and interlobar artery was increased. 叶间动脉舒张期末血流速度 (Vd)的减慢、血流峰速加速时间的延长是高血压病患者最早出现的肾血流动力学异常 ;
The effects of 4 week treatment with tetrandrine (200mg3/d,po) and nimodipine (40mg3/d,po) on renal blood flow (RBF) and glomerular filtration rate (GFR) were studied in 30 patients with essential hypertension,in single blind,random and cross-over trial. 采用单盲、随机和交叉的方法，观察粉防己碱（200mg3/d，po，4w）和尼莫地平（40mg3/d，po，4w）对30例原发性高血压患者的肾血流动力学的影响。
After renal ischemia reperfusion, the high expression of iNOSmRNA in tubules is an important molecular mechanism of renal ischemia reperfusion injury (2) Keep cNOS/iNOS constant may be important for mudulation of renal blood flow and GFR. 缺血再灌注后 ,皮质肾小管上皮中iNOSmRNA的高表达是导致肾缺血再灌注损伤的重要分子机制。 (2 )cNOS/iN OS比值与缺血再灌注过程中肾功能的变化密切相关 ,该比值的恒定对肾血流量和肾小球滤过率(GFR)的调节可能具有重要意义
Reduced RFV/Co reflected the degree of renal injury.3.Many clinical factors affected the change of renal blood flow dynamics such as Co, hematocrit (HCT), and metabolic acidosis. 4. 3.;心输出量的大小、血液粘滞度及祛斑是否合并代谢性酸中毒将对肾血流动力学改变有直接影响。
High serum uric acid resulted in renal function letdown or descending in renal blood flow is more frequent in the patients with hypertension than those with normal blood pressure[1]. 由于高血压病人肾功能减退或肾血流量下降，故高血压患者中高尿酸血症较正常血压者多[1]。
METHODS Using one kidney one clip renal hypertensive model, we determined the arterial pres-sure, heart weight, renal blood flow (RBF) and glomerular filtration rate (GFR) at varied treatment. 方法采用一肾一包扎型肾性高血压模型 ;测定不同条件下动物血压、心脏重量、肾血流量(RBF)和肾小球滤过率 (GFR) .
METHOD: Rabbit models with hemorrhagic shock and Smao shock were used, and the influences of drug on pressure, renal blood flow, lipid peroxide and cell ultrastructure of both models were observed. 方法采用家兔失血性休克模型及肠系膜上动脉夹闭型休克模型,观察药物对休克模型动物的血压、肾血流量、脂质过氧化物、细胞超微结构等的影响。
The changes of systolic peak velocity (VS), diastolic peak velocity (VD), mean glow velocity (VM), pulsatility index (PI) and resistent index (RI) of renal blood flow were observed. 并观测肾血流收缩期峰值流速(VS)，舒张末期峰值流速(VD)，平均流速(VM)，搏动指数(PI)及阻力指数(RI)的变化。
Dynamic renal imaging studies were performed in 132 patients using 99m Tc DTPA and 131 I OIH. Effective renal plasma flow(ERPF) and glomerular filtration rate(GFR) was obtained. 采用 99m Tc- DTPA和 1 31 I- OIH作显像剂 ,对 132例患者行肾动态显像和肾小球滤过率 (GFR)及有效肾血浆流量(ERPF)测定。
Keywords noradrenaline;renal blood flow;angiotonics; 去甲肾上腺素;肾脏血流;血管收缩剂;