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- Results There are many pseudopodia and microvilli on the glioma cell sur face. 结果胶质瘤细胞表面有大量伪足结构和微绒毛结构。
- Results NECL1 was abrogated or markedly reduced in 6 glioma cell lines. 结果 NECL1在6株人神经胶质瘤细胞系中的表达明显下降或缺失。
- Methods Rat glioma cells (D74/HveC) were implanted intracranially in immune-competent rats. 越来越多的研究认为肿瘤微环境是肿瘤生长和治疗反应的决定因素。
- Conclusion Triethyltin chloride can inhibit the proliferation of cultured rat C6 glioma cells and induce its apoptosis. 结论氯化三乙基锡可抑制体外培养C6胶质瘤细胞增殖并诱导细胞凋亡。
- Results BSO depleted the GSH content of glioma cells effectively and increased the radiation sensitivity. 结果经BSO作用后胶质瘤细胞的GSH含量显著下降，放射敏感性增加。
- Abstract: Objective To investigate the invasion ability and collagenolytic activity of human glioma cells in vitro. 文章摘要: 目的：观察人胶质瘤细胞的体外侵袭能力及对胶原的溶解作用。
- Objective: To study the effect and mechanism of morphine on proliferation of rat C6 glioma cells. 目的:研究吗啡对培养的大鼠胶质细胞瘤C6的增殖抑制作用及其机制。
- Abstract Objective To explore the establishment for orthotopic implantation model of human glioma cells in nude mice. 摘要 目的 探讨人脑胶质瘤细胞裸鼠原位移植动物模型建立的技术方法。
- The pseudopodia and microvilli on the glioma cell surface are associated with the invasive ability of the glioma cells. 胶质瘤的微绒毛和伪足结构与其侵袭性相关。
- Objective To study the inhibitory effect of triethyltin chloride on the proliferation of cultured rat C6 glioma cells in vitro. 目的探讨氯化三乙基锡对体外培养大鼠C6胶质瘤细胞的抑瘤作用。
- Objective To investigate the expression level of circadian gene in C6 glioma cells by searching for a novel rhythm-inductor in vitro. 目的寻找一种新的体外节律诱导剂以研究C6神经胶质瘤细胞近日节律基因的表达情况。
- TETC inhibits the proliferation of cultured rat C6 glioma cells in vitro, the mechanism of which may be involved in cell cycle arrest of the cells. TETC可抑制体外培养大鼠C6胶质瘤细胞增殖,其机制可能与C6细胞周期阻滞在G0/G1期有关。
- MMC has cytotoxicity effect on cultured rat C6 glioma cells in vitro,and can inhibit proliferation and induce apoptosis. MMC能够杀伤大鼠C6胶质瘤细胞,抑制增殖,诱导凋亡,具有应用于胶质瘤治疗的潜在价值。
- The establishment of the gene expression profile and cloning the genes associated with differentiation inducing of human glioma cells. 人脑胶质瘤细胞诱导分化基因谱的建立及相关基因的克隆
- In this research, we used an in vitro BBB model to investigate the impacts of glioma cells on the blood-brain barrier and their molecular mechanisms. 为了探索胶质瘤细胞对血脑屏障的多种作用及其分子机制，本研究通过建立体外血脑屏障模型，探索了胶质瘤细胞对血脑屏障的多种作用及其可能的分子生物学与病理生理学机制。
- The inhibition rate of C6 glioma cells at different times(6,12,24,48 and 72 h)after SDT treatment was investigated by MTT assay. 采用噻唑蓝比色分析法(MTT法)观察SDT处理后6、12、24、48和72h的抑瘤率；
- NaPA not only inhibit the growth of P 168 haman glioma cells, but also remarkably induce the differentiation of these cells in vitro. NaPA在体外不仅能够抑制人胶质瘤细胞的生长 ,而且对肿瘤细胞有明显的诱导分化作用
- OBJECTIVE: To explore the mechanism of killing tumor cells by observing the C6 rat glioma cell apoptosis after cryotherapy. 摘要目的：通过观察大鼠C6脑胶质瘤经冷冻后出现细胞凋亡，探讨冷冻对肿瘤的杀伤机制。
- Haase H,Watjen W,Beyersmann D.Zinc induces apoptosis that can be suppressed by lanthanum in C6 rat glioma cells[J].Biol Chem,2001,382(8):1227. 肖白;纪云晶;崔明珍;等.;镧铈对癌细胞恶性增殖及相关基因表达的影响[J]
- Conclusion Rats immunized with HAC purified from C6 cells develop immuno resistance against C6 glioma cells, and their survival rates are improved. 结论 C6细胞中HAC可以诱导大鼠产生对C6细胞的细胞免疫，提高大鼠存活率。
