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- mitochondria endopeptidase 线粒体内肽酶
- Mitochondria do not use the universal genetic code. 线粒体并不使用通用的遗传密码。
- Kallikrein is a kind of endopeptidase. 激肽释放酶是一种内切蛋白酶。
- Mitochondria are present in all eukaryotic cells. 一切真核的细胞都有线粒体。
- They have also been isolated from mitochondria. 目前已从线粒体中分离出铁硫蛋白。
- They thus cut their mitochondria out of the loop. 这种作用机制效率较低，但不需要线粒体参与。
- But what makes mitochondria lose steam? 但是什么导致线粒体失效呢？
- Mitochondria are tiny energy units within eery cell of the body. 在身体每个细胞中线粒体是很小的能量单位。
- The midpiece consists of centrioks, mitochondria and ribosomes. 中段由中心粒、线粒体和核糖体组成;
- "Mitochondria are guardians of cell survival," said Sinclair. “线粒体保证细胞存活。”
- The mitochondria are well developed with numerous cristae. 外层则连续迴肠细胞。
- The stress forces mitochondria to operate more efficiently. 这种压力迫使线粒体提升效率，产生更多能量。
- Mitofusin 2 tethers endoplasmic reticulum to mitochondria. 线粒体融合蛋白2把内质网连接到线粒体。
- The oxaloacetate level in the mitochondria is determined by the redox state. 线粒体中草酰乙酸水平又取决于氧化还原状态。
- From TEM ,we can see Mitochondria and RER was reduced by high concentration NO. 电镜观察可见实验1)、2)、3)组线粒体减少，粗面内质网减少，凋亡细胞增多。
- Bcl-2 protein immunoreactivity was associated with mitochondria and ER. Bcl 2分布与线粒体和内质网有关。
- The dysfunction of mitochondria involves in the pathogenesis of shock and MODS. 线粒体功能紊乱参与了休克和多器官功能不全的发生。
- Enzymes and pathways found in various manifestations of mitochondria. 表明,明显,现象,表现,表示,显示
- Objective To investigate the expression of substance P (SP), neurokinin-1 receptor (NK-1R) and neutral endopeptidase (NEP) in human pancreatic cancer. 目的 了解P物质(SP)、神经激肽-1受体(NK-1R)及中性肽链内切酶(NEP)在胰腺癌细胞中表达。
