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- Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC). 目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。
- The renal tubular cells appear foamy because of the accumulation of neutral fats and mucopolysaccharides. 因为中性脂肪和粘多糖堆积，肾小管细胞呈泡沫样。
- Abstract： Objective To study the cytopathogcnic effect of epidemic hemorrhagic fever with renal syndrome virus(HFRSV) on renal tubular cells(RTC) . 摘 要： 目的研究肾综合征出血热病毒(HFRSV)对肾小管上皮细胞(RTC)的致病作用。
- Objective To observe the status of aggregation of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on infected human fetal renal tubular cells (RTC) and the effects of Arg Gly Asp peptides(RGD). 目的 观察人胚肾小管上皮细胞 (RTC)受肾综合征出血热病毒 (HFRSV)感染后粘附聚集状态的改变和精氨酸 甘氨酸 天门冬氨酸序列 (Arg Gly Asp ,RGD)对这一改变的影响。
- Methods In vitro transfered and cultured renal tubular cells AT2 receptor agonist CGP - 42112A in different doses were diffused, then cell apoptosis was analyzed by TUNEL and flow cytometry. 方法 通过取新生大鼠肾小管上皮细胞体外培养,传代后加入不同浓度AT_2受体兴奋剂CGP-42112A,在不同时间用流式细胞仪测定凋亡细胞比值,并结合TUNEL分析。
- However, these effects of SIR could be inhibited by PDS.Conclusions： The membrane stability of cultured renal tubular cells injured by theserum of renal IR could be promoted by Panaxadiol Saponins. 结论：PDS具有增强急性肾缺血再灌流性免血清致伤的培养人肾小管细胞膜稳定性的作用。
- STUDY ON THE EFFECT AND MECHANISM OF IL - IRA ON CULTURED HUMAN RENAL TUBULAR CELLS IL-1受体拮抗剂对培养人肾小管细胞的影响及机制研究
- Keywords Panaxadiol saponins;renal tubular cells;perioxide reaction;proliferation; 人参二醇组皂甙;肾小管细胞;脂质过氧化;增殖;
- Study on transdifferentiation of renal tubular cells in rat chronic renal interstitial fibrosis induced by Radix Aristolochiae Fangchi extract 广防己提取物致大鼠慢性肾间质纤维化的机制研究
- Keywords Panaxadiol saponins The rabbit serum of acute renal ischemic reperfusion Culture Human renal tubular cell; 关键词人参二醇组皂甙;肾缺血再灌流性兔血清;培养;肾小管细胞;
- renal tubular cells 肾小管上皮细胞
- renal tubular cell 肾小管细胞
- Renal tubular cell (RTC) 肾小管上皮细胞
- Alternations of ADF and G-actin in Renal Tubular Epithelial Cell of Neonatal Rats During Kidney Ischemic Injury. 新生大鼠肾脏缺血性损伤致肾小管上皮细胞ADF与G-ACTIN的改变
- The Bence-Jones protein (BJP) in the urine of patients with multiple myeloma (MM) shows direct toxicity on the renal proximal tubular cells (PTC). 多发性骨髓瘤(MM)患者尿本周蛋白(BJP)对肾近端小管上皮细胞(PTC)具有直接毒性,本研究通过体外观察BJP对PTC的凋亡作用以探讨BJP对PTC的部分毒性机制。
- Cell adhesion has a vital effect on renal inflammatory injury, renal tubular lesions and interstitial fibrosis. 其在肾脏炎性病变、肾小管损伤、肾间质纤维化等发生发展中起重要作用。
- Renal interstitium was hyperemic, the glomerular volume became bigger, and renal tubular epithelial cells showed swelling and glassy degeneration. 肾间质充血，肾小球体积增大，近曲小管上皮细胞肿胀、玻璃样变。
- The renal biopsy showed features of acute tubular necrosis, deposition of hemosiderin and positive urate stain in the proximal tubular cells, without vascular thrombosis. 肾脏切片检查显示急性肾小管坏死，血铁质沉积于近端肾小管细胞并且呈阳性尿酸盐染色，无血管栓塞。
- Although the degree of apoptosis in tubular cells correlates with serum creatinine in proliferatie GN at the time of biopsy, it is of limited use to predict future renal function. 尽管肾小管细胞凋亡程度与同期活检的增生性肾小球肾炎的血肌肝相关，但其对于预测未来肾功能作用仍有限。
- Objective To establish sublytic membrane attack complex (sMAC) model and to identify the sublytic effects on renal tubular epithelial cell(TEC). 目的体外组装补体膜攻击复合物(membrane attack complex,MAC),建立肾小管上皮细胞HK-2亚溶破模型,为进一步探讨小管间质的免疫性损伤效应奠定基础。
