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- Keywords secondary neuronal injury apoptosis intro-ventricle hypothermia(IVH); 继发性神经元损伤;细胞凋亡;脑室内低温;
- secondary neuronal injury 继发性神经元损伤
- At this point, the secondary neuronal axons decussate and continue to ascend as the medial leminiscus. 在一级和二级感觉神经元之间形成突触的地点,二级神经元轴突将如内侧丘系一般交叉上升。
- Brain-derived neurotrophic factor (BDNF) was shown to induce antiapoptotic mechanisms after stroke and to reduce infarct size and secondary neuronal cell death. 脑源性神经营养因子(BDNF)被显示了卒中后可诱导抗凋亡机制并减少梗塞面积以及继发神经细胞死亡。
- Brain-deried neurotrophic factor (BDNF) was shown to induce antiapoptotic mechanisms after stroke and to reduce infarct size and secondary neuronal cell death. 脑源性神经营养因子(BDNF)被显示了卒中后可诱导抗凋亡机制并减少梗塞面积以及继发神经细胞死亡。
- Conclusion:NGF has a protective effect on neuronal injury induced by reperfusion postischemia. 结论:NGF对缺血再灌注所致的神经细胞损伤可能具有一定的保护作用。
- Hypoxia may usually cause an increase in blood-brain barrier (BBB) permeability, resulting in vasogenic brain edema and aggravating neuronal injury. 缺氧通常会引起血脑屏障通透性增高,造成血管源性脑水肿加重神经元损伤。
- White B,Sulliran JM,De Gracia DJ,et al.Brain ischemia and reperfusion, molecular mechanisms of neuronal injury[J].J Neurol Sci,2000,179:1. 张世仪刘洁.;山莨菪碱对实验性脑损害所致认知障碍的改善作用[J]
- Therefore, it may be an important approach to treating ischemic neuron injury by fullyinducing the expression of endogenous NTFs and their receptors. 所以,充分诱导内源性NTFs 及其受体的表达,可能是治疗缺血神经元损伤的重要途径。
- AIM: To explore the effects of carbenoxolone (CBX), a gap junction blocker, on the neuron injury induced by focal cerebral infarct. 目的 :探讨缝隙连接阻断剂甘珀酸对大鼠光化学局灶性脑梗死神经元损害的影响 .
- Wang KW, Yang ZH, Wang ZG, et al.Expression of C-fos proteinafter cultured cortical neuron injury in rats [J].Chin J Traumatol, 1998, 14(4): 219-22. [2]王克万;尹志勇;杨志焕;等.;培养大鼠皮层神经元损伤后c-fos蛋白表达[J]
- Purpose To study the effects and the possible mechanisms of Choerospondias axillaries fruit (CAF) on rat cultured cortex neuron injury induced by Serum-Free DMEM. 摘要目的研究广枣及其提取组分对拟衰老神经细胞的保护作用并初步探讨其机制。
- AIM: To explore the mechanism of neuronal injury and repair by investigating the expression of caspase-3 and apurinic/apyrimidinic endonuclease (APE/Ref-1) after focal cerebral ischemia. 目的 :观察脑缺血后半胱氨酸蛋白酶 3 (caspase - 3 )和脱嘌呤 /脱嘧啶核酸内切酶 (APE/Ref- 1)的表达 ,探讨脑缺血损伤与修复机制。
- Aim: To observe the effects of lidocaine and thiopental on the neuronal injury induced by the experimental ischemia in hippocampus slice cultures obtained from postnatal 22 days SD rats. 目的:观察利多卡因和硫喷妥钠对生后22d大鼠培养海马脑片的实验型缺血后神经元损伤的影响。
- After synapsing, the secondary neurons decussate and ascend as the spinothalamic tract in the anterior lateral portion of the spinal cord. 在建立突触后,二级感觉神经元将如脊髓前外侧部分中的脊髓丘脑束般交叉上升。
- Kapinya KJ,Lowl D,Futterer C,et al.Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent[J].Stroke,2002,33(7):1889. 李继庆;张联峰;王洋松;等.;异氟醚预处理对大鼠全脑缺血再灌注损伤的保护作用[J]
- When AMPA receptor is activated,lots of intracellular Ca~(2+)result in neurons injury. 在AMPA受体激活时,大量Ca2+内流导致神经损伤。
- As the primary axons reach the caudal medulla, they leave their respective fasiculi and enter and synapse on secondary neurons within the nucleus gracilis and the nucleus cuneatus, respectively. 一级感觉神经元的轴突会伸延至延髓的尾部,且拥有细长细胞核或楔形细胞核的神经元会分别透过进入或于二级感觉神经元及轴突间形成突触而离开延髓的尾部。
- These results imply that extrinsic NAD+ prevented effectively the neuronal injuries caused by OGD treatment through enhancing the ability of repairing endogenous oxidative DNA damage. 4. 结果提示NAD+通过提高内源性DNA修复能力,有效地抵抗了OGD引起的神经元DNA损伤。
- Her age is of secondary interest. 她的年龄不太重要。