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However,Bromocriptine did not alter systemic vascular resistance. 氟呱啶醇能阻断溴隐亭这一作用；
There was a tendency for the pulmonary vascular resistance to decrease in HS and HSD groups as compared with NS group. 与NS组比较,HS组和HSD治疗后肺血管阻力呈下降趋势。
Arteriolar relaxation reduces systemic vascular resistance and arterial pressure(afterload). 心脏指数可增加、降低或不变。
Significantly increase in coronary blood flow due to reduction of vascular resistance. 可增加冠状动脉流量，因为丹参能减低血管阻力。
There was no difference of other parameters [cerebral vascular resistance (CVR), compliance, Co and CP] in every group (P>0.05). 其他各项参数(脑血管阻力、顺应性、零压顺应性、临界压力)各组间无差异(P>0．05)。
Results and Conclusion: Inhaled iloprost can reduce pulmonary vascular resistance and delay the development of primary pulmonary hypertension. 结果和结伦：吸入性伊洛前列素治疗可以降低肺血管阻力和延缓原发性肺动脉高压的进程。
Diltiazem is a benzothiazepine derivative calcium antagonist，which produces antihypertensive effect primarily by relaxation of vascular resistance. 地尔硫卓是一种苯噻氮卓类钙拮抗剂，作用于血管平滑肌使外周血管扩张，心脏后负荷降低，发挥降压作用。
With prazosin, the average cardiac index increased (by) 20 percent and systemic vascular resistance decreased (by) 20 percent. 使用哌唑嗪，平均心脏指数增加20%25，全身血管阻力下降20%25。
Conclusion: Epidural anesthesia increase umbilical blood flow，but does not change the umbilical vascular resistance, which is benefical for foetus. 结论：硬膜外阻滞后可增加脐血流而不增加脐血管阻力，对胎儿血供是有利的。
Results and Conclusion:Inhaled iloprost can reduce pulmonary vascular resistance and delay the development of primary pulmonary hypertension. 结果和结论:吸入性伊洛前列素治疗可以降低肺血管阻力和延缓原发性肺动脉高压的进程。
Introduction: Reperfusion syndrome in liver transplant patients is characterized by labile systemic vascular resistance (SVR) and cardiac output (CO). 简介：肝移植患者发生再灌注综合征的特征是体循环阻力（SVR）不稳定和心输出量（CO）不稳定。
Endothelin-1 contributes to increased systemic vascular resistance,vascular remoulding,left ventricular dysfunction and renal impairment in essential hypertension. 原发性高血压时血浆内皮素-1水平升高,可导致外周阻力增加,并且通过激活ETA和ETB受体可加重高血压病心、肾、动脉系统的损害,形成恶性循环。
RESULTS &CONCLUSION: The feature of PAH is that pulmonary vascular resistance progressively increases,and finally leads to right heart failure and death. 结果与结论:肺动脉高压的特点是肺血管阻力进行性增加,最终导致右心衰竭和死亡。
Different forms of PH are characterized by pronounced vascular remodeling, resulting in increased vascular resistance and subsequent right heart failure. 各种肺动脉高压形成都是以血管显著的重构为特征，从而导致血管阻力增加，随之出现右心衰。
RPC significantly reduced mean artery pressure (MAP),systemic vascular resistance (SVR) and coronary vascular resistance (CVR,P<0.05) and slightly increased cardiac output(CO) and coronary blood flow (CBF). RPC显著降低平均动脉压(MAP)、外周血管阻力 (SVR)及冠脉循环阻力 (CVR) (P<0 .;0 5 ) ; 轻度增加心排出量及冠脉血流量 (P>0
The cardiac output (CO) and Cst decreased significantly when the PEEP was 20 cm H 2O, at the same time, the pulmonary vascular resistance (PVR) and the systemic vascular resistance (SVR) increased (P<0.05). 在PEEP增加到 2 0cmH2 O时心输出量 (CO)和Cst下降 ;外周循环阻力 (SVR)、肺循环阻力 (PVR)和气道峰值压 (PIP)增加 (P <0 .;0 5 )。
The responses to electrical stimulation of VSMp showed a rise of AP(44.49%,P< 0.001),vascular resistance of muscles(VRm 37.08%,P<0.001)and vascular resistance of kidney (VRk 26.59,P<0.01). 肾血管阻抗上升26.;5%25(P<0
By contrast, hydralazine increased cardiac index by 58 percent and decreased systemic vascular resistence by 40 percent. 相比之下，使用肼苯达嗪，心脏指数增加58%25，而全身血管阻力减少40%25。
The results were as follows:1.The mean pulmonary arte-rial pressure(P pa)and pulmonary vascular resistance(PVR)were signiflcantly decreased by in-haled NO in chronic hypoxic rats(P<0.01),but these changes were not observed in normoxiccontrol rats. 结果表明：（1）常氧吸入NO时能明显降低慢性缺氧大鼠肺动脉平均压（Ppa）和肺血管阻力（PVR），但对正常大鼠的Ppa和PVR无明显影响；
Methods Coronary blood flow (CBF) intra-arterial blood pressure, heart rate, and coronary vascular resistance (CVR) were measured at rest, during isometric exercise before and 60 min after Losartan (25 mg/kg, ig). 结果发现 ,Losartan明显降低高血压大鼠的平均动脉压和冠状动脉阻力 ,明显增加其冠状动脉血流量 ,而对其心率无明显影响 ;