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Kupffer cell medium 枯否细胞介质
There is lobular disarray with focal hepatocyte necrosis, giant cell transformation, lymphocytic infiltration, Kupffer cell hyperplasia, and cholestasis (not seen here). 图中可见具有病灶肝细胞坏死、巨细胞转化、淋巴细胞浸润、枯否细胞增生和胆汁淤积（此图未显示）的肝小叶紊乱。
On the contrary, the prominent features in liver section of the RC group were inflammatory cell (notably mononuclear) infiltration and Kupffer cell activation. RC组炎症反应较突出，主要为单核细胞浸润以及枯否细胞活跃，大滋养体和裂殖体在肝血窦滞留；
Furthermore, TG1-1 cell medium supplemented with estrogen-induced BM-EPC mediated tubulogenesis, which was an experimental in vivo representation of the neovasculature. 细胞水平的分析显示雌二醇可以刺激骨髓源性内皮细胞祖细胞通过旁分泌作用刺激TG1-1增生和迁移。
The hepatocytes and Kupffer cells here are full of granular brown deposits of hemosiderin from accumulation of excess iron in the liver. 图中肝细胞和枯否细胞内充满由肝脏中过多的铁积聚造成的含铁血黄素的褐色粒状沉着。
Objective To observe the synthesis of TLR2 protein and mRNA expression in Kupffer cells(KCs). 目的观察小鼠肝部分缺血 /再灌注损伤时肝脏Kupffer细胞表面TLR2mRNA的表达及蛋白质的合成情况。
A Prussian blue iron stain demonstrates the blue granules of hemosiderin in hepatocytes and Kupffer cells. 普鲁士蓝铁染色显示肝细胞和枯否细胞中含铁血黄素的蓝色颗粒。
Oligodendrocyte Precursor Cell Medium (OPCM) is a complete medium designed for optimal growth of normal human oligodendrocyte precursor cells in vitro. 少突胶质前体细胞培养基是专门为正常人类少突胶质前体细胞体外培养设计的最适于其生长的培养基。
Prussian blue iron stain demonstrates the blue granules of hemosiderin in hepatocytes and Kupffer cells. 普鲁士蓝铁染色显示肝细胞和枯否细胞中含铁血黄素的蓝色颗粒。
Mesenchymal Stem Cell Medium (MSCM) is a complete medium designed for optimal growth of normal human mesenchymal stem cells in vitro. 间充质干细胞培养基是专门为人类间充质干细胞体外培养所设计的、最适合其生长的培养基。
Meningeal Cell Medium (MCM) is a complete medium designed for optimal growth of normal human brain derived meningeal cells in vitro. 脑膜细胞培养基是专门为正常人类脑膜细胞体外培养设计的最适于其生长的培养基。
Many erythrocyte and kupffer cells were seen in sinusoid of liver and spleen, along with many yellow and brown irons contained blood grains. 大量肝细胞肿胀，胞浆内可见许多大小不一的脂肪空泡；肝细胞胞核浓缩、碎裂、溶解甚至消失。
CONCLUSION Liver fibrosis was related to the proliferation of both Kupffer cells and lipocytes and their expression of bFGF. 主题词肝硬变，实验性/代谢；成纤维细胞生长因子，碱性/代谢；免疫组织化学
Urothelila Cell Medium (UCM) is a complete medium designed for optimal growth of normal human urothelial cells in vitro. 尿道上皮细胞培养基是专门为正常人类尿道上皮细胞体外培养设计的最适于其生长的培养基。
Smooth Muscle Cell Medium (SMCM) is a complete medium designed for optimal growth of normal human smooth muscle cells in vitro. 内皮细胞培养基是专门为正常人类微血管内皮细胞体外培养设计的最适于其生长的完全培养基。
A Prussian blue reaction is seen in this iron stain of the liver to demonstrate large amounts of hemosiderin that are present in hepatocytes and Kupffer cells. 肝脏普鲁士蓝染色显示,呈蓝色的含铁血黄素颗粒大量沉着在肝实质细胞和。
Prussian blue reaction is seen in this iron stain of the liver to demonstrate large amounts of hemosiderin that are present in hepatocytes and Kupffer cells. 肝脏普鲁士蓝染色呈蓝色的含铁血黄素颗粒大量沉着在肝实质细胞和库普弗细胞内。
Cirrhotic Kupffer cells also had increased densities of functional receptors for both PAF and ET-1(exclusively ETB),but did not change the affinity of these receptors. No mRNA transcripts for the ETA receptor or preproET-1 were detected. 肝硬化组Kupffer细胞PAF及ETB受体mRNA水平及受体密度明显增加,而受体亲和力无变化,无ETA受体和ppET-1 mRNA表达。
Objective:To observe the synthesis of Toll-like receptor (TLR)4 protein and its mRNA expression in Kupffer cells (KCs) and evaluate the role of TLR 4 in the liver injury in the rats with alcohol-induced liver disease (ALD). 目的 :观察酒精性肝病大鼠Kupffer细胞 (KCs)Toll样受体 (TLR) 4蛋白合成和基因表达及其在肝损害中的作用。
Thus,the aim of this study was to characterize the effect on the antigen-presenting function of Kupffer cells by intrasplenic transplantation of GM-CSF gene-modified fetal liver cells (FLC-GM)in combination with IL-2 and 5-Fu. 方法 :以结肠癌肝转移小鼠为模型 ,以脾内移植GM CSF基因修饰的胎肝细胞 (FLC GM)与低剂量IL 2、5 Fu腹腔注射作为基因治疗与化学免疫治疗联合应用方案。