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- The physiology character of cardiac muscle includes stimulant, autonomic sex, conductibility and contractility. 心肌的生理特性包括兴奋性，自律性，传导性和收缩性。
- Ultrastructure of cardiac muscle cell such as mitochondrion and cardiac muscle fibers was atttenuated. 明显延缓心肌细胞线粒体、心肌纤维细胞的病理改变。
- Image Analysis of Cardiac Muscle Cytoskeleton under Simulated Microgravity Based on Gray-level Co-occurrence Matrix(GLCM). 基于灰度共生矩阵特征的对模拟微重力条件下心肌细胞骨架的图像分析
- Magnetism resonance can be ticked off delimit clear-cut heart each room, room is removed, heart valve reachs the image of cardiac muscle. 磁共振可勾划轮廓清楚的心脏各房、室间隔，心瓣膜及心肌的图像。
- Methods: The fresh tissues of cardiac muscle sleeve in superior and inferior vena cava were taken from goat hearts and sliced up in routine methods. 方法：取新鲜山羊上下腔静脉的肌袖组织，常规组织切片，光镜和电镜观察及细胞计量。
- Action and utility: Enhance power of cardiac muscle systole, outspread coronary artery, decelerate rhythm of the heart, have diuresis effect. 作用与用途：增强心肌收缩力，扩张冠状动脉，减慢心律，并有利尿作用。
- ObjectiveTo study the effect of ulinastatin on perioperative zymogram of cardiac muscle in conventional coronary artery bypass. 目的研究乌司他丁对体外循环下冠脉搭桥术患者术后期心肌酶谱的影响。
- Then, test Na+_K+_ATPase,Mg++_ATPase,Ca++_ATPase,Ca++_Mg++_ATPase of the membrane of cardiac muscle cell of four groups of experimental animals and the blood culture. 测定四组实验动物心肌细胞膜上Na +_K + -ATPase,Mg+ + -ATPase,Ca + +_ATPase,Ca + +_Mg+ + -ATPase4种ATP酶的活力 ,同时作血培养。
- The chroma difference of Potassium of inside and outside of cardiac muscle cell is systole of cardiac muscle fiber - the premise condition that diastole moves. 心肌细胞内外钾的浓度差是心肌纤维收缩-舒张运动的前提条件。
- Objective To analyze of efficacy of combination of polarized solution of Ka and Mg with Betaloc for treatment of cardiac muscle toxicity caused by Adriamycin. 摘要目的分析倍他洛克加钾镁极化液预防阿霉素的心脏毒性的效果。
- Coronary heart disease(CHD) is a type of cardiopathy that depends on atherogenesis of coronary artery and that induces the ischemia and anoxia of cardiac muscle. 冠心病是指冠状动脉粥样硬化使冠状动脉管腔狭窄,导致心肌缺血、缺氧而引起的心脏病,是危害人类健康的常见病、多发病。
- The mechanism may be related with increasing SOD activity and enhancing the antioxidation property and stabilizing the cell transmembrane potential of cardiac muscle. 结论：黄连素预处理对大鼠心肌缺血再灌注损伤具有保护作用，其机制可能与增加SOD活性、增强心肌抗氧化能力、稳定心肌细胞膜电位有关。
- The Branched fibres of cardiac muscle give it a netlike structure; contraction originates in the heart's muscle tissue itself with a signal from the natural pacemaker; vagus and sympathetic nerves control heart rate. 心肌树枝状的纤维就像网状结构;收缩是源自于心脏肌肉组织本身,由天然的节律点产生讯号;迷走神经与交感神经控制心脏的跳动速率。
- The number of cardiac muscle cell apoptosis was higher in CLP than the control group(p<0.01) . The number of cardiac muscle cell apoptosis at 2h and 3h was higher than at 4h and 5h, there was a noble discrepancy (p<0.05). CLP术后各时限组心肌细胞凋亡数目均高于对照组;且均有显著性差异(P<0.;01);但2h组、3h组心肌细胞凋亡数目高于4h组、5h组;有统计学差异(P<0
- Cx40,Cx43,Cx45,Cx37 and Cx46.Among them Cx40,Cx43,Cx45 is relatively abundant in cardiac muscle,Cx37 distributing to wall of cardiac vascular,a small quantity of Cx37 and Cx46 distributing to atrium or ventricle. 在心房或心室肌细胞中偶尔可发现微量的Cx37、Cx46。 Cx43除了窦房结、房室结、部分传导系统外，存在于整个心脏，Cx40表达在心房(小鼠心脏除外)和传导系统近端最强，Cx45局限于窦房结、房室结及传导系统。
- In addition, still can keep clear of the insulin when heart failure is secreted suffer check status, cardiac muscle metabolization gets ameliorative, enhance power of cardiac muscle systole thereby. 此外，还可清除心力衰竭时胰岛素分泌受抑制状态，心肌代谢得到改善，从而加强心肌收缩力。
- Arrhythmogenic right ventricular cardiomyopathy (ARVC) is one kind of cardiac muscle disease that the cardiac muscle of right ventricular is replaced by fibre and/or fat with unknown cause. 致心律不齐性右室心肌病是以右室心肌被纤维脂肪取代为特征的一种原因不明的心肌病,通常表现为局限性右室病变。
- These are signs of cardiac arrest. 这就是心博停止的迹象。
- conductivity of cardiac muscle cells 心肌细胞传导性
- excitability of cardiac muscle cells 心肌细胞兴奋性
