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Brain ischemic disease 脑缺血性疾病
Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
These processes are observed during ontogenesis, tumor progression, ischemic disease or physical training. 这些过程可在个体发育，肿瘤进展，局部缺血性疾病或体检中发现。
These processes are obsered during ontogenesis, tumor progression, ischemic disease or physical training. 这些过程可在个体发育，肿瘤进展，局部缺血性疾病或体检中发现。
Both Brain ischemic preprocessing and postprocessing can result in neuroprotective effect by inducing brain ischemic tolerance. 脑缺血预处理及后处理均可以诱导脑缺血耐受形成而产生神经保护作用。
DWI is sensitive to early ischemic changes,which can find the changes of brain ischemia earlier than that of routine T2WI. DWI对早期缺血改变非常敏感,DWI较常规T2加权图像能更早地发现脑缺血的改变。
Conclusion: Venous arterialization could improve the blood supplying of the limbs and is an effective surgical protocol treat ischemic disease of the lower extremity. 结论：静脉动脉化手术可以改善肢体远端血供，是治疗下肢动脉广泛闭塞较有效的方法。
Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
Objective To analyze and summarize diagnose of mesenteric ischemia disease and the treatment effect. 目的分析总结肠系膜缺血性疾病的诊断及治疗效果。
But the detailed mechanisms of G-LOC or G induced brain ischemia as well as protective effect IPC were not clear. 然而不论大G昏迷、G力造成的脑损伤及缺血前制约效应的保护机转都并不清楚。
It can cause severe complications, such as brain ischemia or cervical hematorrhea incurred by rupture of aneurysm. 它可以引起严重的并发症，比如大脑局部缺血或动脉瘤破裂产生的颈部软组织出血。
It was suggested that DDPH had some protective effect on acute brain ischemia and ischemia-reperfusion. 结果提示，DDPH对动物脑缺血具有一定的保护作用。
The pathologic changes of RPE cells is of very importance to the development prosess of retinal hypoxia and ischemia disease. 在视网膜缺血缺氧性疾病中，RPE细胞的异常病理变化在疾病的发生发展过程中占重要地位，探讨RPE细胞在缺氧应激这一病理情况下的异常生物学行为可能会为这类疾病的发病机制、预防和防治奠定理论基础。
Abstract: Brain ischemic preconditioning is that transient ischemia in sublethal dose will result in tolerance to the following ischemia in lethal dose for long time. 摘要：脑缺血预处理即给予短暂亚致死量缺血可对随后的长时间致死性缺血损伤产生耐受。
Objective To study the improvement effects of GM_1 on brain ischemia reperfusion(BIR) induced memory impairment in mice. 目的探讨脑缺血复灌后连续应用GM1对改善记忆障碍的疗效。
Disturbances of calcium homeostasis and modification of ca~(2+) pump(Ca~(22+),Mg~(2+)-ATPase)in the rabbit focal brain ischemia. 脑缺血时钙稳态与钙泵(Ca~(2+),Mg~(2+)-ATP酶)活性变化
Endothelial progenitor cells (EPC) play a key role in the adult vasculogenesis.The discoveries of EPC provide a new therapy for the ischemic disease which could not be treated by revascularization. 血管内皮祖细胞（EPC，或称血管内皮前体细胞）在促进成体血管新生方面起着关键作用，它的发现为无法依赖再血管化治疗的缺血性疾病提供了一种新的治疗方法。
Without obvious influence in brain metabolic functions,L1 KO mice is an useful animal model for investigation of the role of L1 in brain ischemia. L1CAM基因缺失对小鼠脑局部代谢功能无明显影响,提示L1 KO小鼠可作为研究L1CAM在脑缺血中作用的动物模型。
The abnormal DA release inhibited by L-NNA during global ischemia may be one of the protective mechanism of this agent on brain ischemia. L-NNA抑制半球缺血时DA的不正常释放可能是其保护缺血大脑的机制之一。