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- The ovarian cancer cell line transfered by HSV TK gene regressed rapidly in response to the GCV therapy. AO细胞转染HSV－TK基因后，能有效地被GCV杀灭。
- Apoptosis of drug-resistant human ovarian cancer cell line COC_1/DDP induced by survivin antisense oligonucleotides. Survivin反义寡核苷酸诱导人卵巢癌耐药细胞株COC_1/DDP凋亡的实验研究
- Objective:To study the mechanism of sodium butyrate (NaB) on human ovarian cancer cell line 3AO. 目的 :研究丁酸钠逆转人卵巢癌 3AO细胞恶性表型的机制。
- The effect of human interleukin 2 gene transfer on the adhesive ability of ovarian cancer cell line SKOV3. IL-2基因转移对卵巢癌细胞系SKOV3粘附性的影响
- Conclusion:The reconstituted SCID mouse model with human ovarian cancer cell line has been established successfully. 结论：免疫重建的荷人卵巢癌-SCID小鼠模型已初步建立成功。
- Survivin antisense oligonucleotides could effectively induce apoptosis of drug-resistant human ovarian cancer cell line COC1/DDP. survivin ASONs反义寡核苷酸能诱导人卵巢癌耐药细胞株COC1/DDP细胞凋亡 ,且效果显著
- Objective:To explore the molecular mechanism of topotecan-induced apoptosis in cisplatin-resistant human ovarian cancer cell line COC1/DDP. 目的探讨拓扑替康(TPT)诱导人卵巢癌顺铂耐药株COC1/DDP凋亡的内在分子机制。
- Objective:To investigate the effects of all-trans retinoic acid (ATRA) on prolifetation and differentiation in ovarian cancer cell line COC1 in vitro. 目的探讨全反式维甲酸(ATRA)对人卵巢癌细胞株COC1增殖分化的影响。
- Objective:To study the effects of tumor suppressor gene mda-7/IL-24 on the ovarian cancer cell line A2780 and drug-resistant cell line A2780/DDP. 目的:探讨抑癌基因mda-7/IL-24对卵巢癌细胞株A2780和耐药株A2780/ DDP耐药性的影响。
- Conclusion ATP7B antisense oligodeoxynucleotide increased the sensitivity of cisplatin in ovarian cancer cell line SKOV3ipl. 结论 ATP7B基因反义寡核苷酸能增加SKOV3ip1细胞对顺铂的敏感性。
- Objective To investigate the effect of lung resistance protein(LRP) on the resistance phenotype of ovarian cancer cell line A2780/DDP to cisplatin and its molecular mechanism. 目的探讨肺耐药蛋白(lung resistance protein,LRP)与人卵巢癌顺铂耐药细胞A2780/DDP顺铂耐药表型的关系及其分子机制。
- Methods: Human ovarian cancer cell line COC1 was cultured in medium containing ATRA in different concentrations in vitro. Cell proliferation inhibition was tested by MTT assay. 方法：选用人卵巢癌细胞株COC1进行体外培养，以不同浓度的ATRA处理COC1细胞，四甲基偶氮唑蓝（MTT）比色法检测细胞增殖抑制情况；
- Methods: The cisplatin-resistant ovarian cancer cell line COC1/DDP was cultured and injected into the abdominal cavity of nude mice to establish the malignant ascites model. 方法：取体外培养的卵巢癌顺铂耐药细胞株COC1/DDP，注射于裸鼠腹腔，建立裸鼠卵巢癌腹水瘤模型。
- Methods: Human ovarian cancer cell line COC1 was cultured in medium containing ATRA in different concentrations in vitro.Cell proliferation inhibition was tested by MTT assay. 方法：选用人卵巢癌细胞株COC1进行体外培养，以不同浓度的ATRA处理COC1细胞，四甲基偶氮唑蓝（MTT）比色法检测细胞增殖抑制情况；
- Abstract Objective ：To observe the effect of cisplatin, taxol and adriamycin on in vitro growth as well as the nature of cell death of human ovarian cancer cell line OVCAR-3. 摘要 目的 ：观察抗卵巢癌药物顺铂、紫杉醇和阿霉素对卵巢癌细胞系OVCAR-3体外生长和生存的影响并分析由它们所致的细胞死亡性质。
- Objective:To evaluate the adhesive ability of ovarian cancer cell line SKOV3 and the IL 2 gene modified cells. 目的：研究IL－2基因转移对卵巢癌SKOV3细胞系粘附性的影响。
- Objective To detect the intracellular cisplatin concentration in both multicellular aggregates and monocellular suspension of ovarian cancer cell line COC1 and its cisplatin-resistant subline COC1/DDP. 目的 了解人卵巢癌细胞株COC1及其耐药亚株COC1/DDP以多细胞团簇形式和单细胞形式存在的细胞内顺铂浓度的差异 ,探讨细胞存在形式在COC1/DDP耐药中的作用。
- Objective To investigate the inhibitory effects of rose roxburghii tratt juice(RRTJ) and morinda citrifolia juice(NoniJ) on the growth of human ovarian cancer cell line COC_2 in vitro. 目的探讨刺梨汁(RRTJ)、诺丽汁(Non iJ)及刺梨汁联合诺丽汁对卵巢癌细胞株COC2体外生长的抑制作用。
- Objective; To investigate the expressions of anti-apoptotic protein Bcl-XL,Bcl-2, cytochrome C and the caspase-3 activity in cisplatin-resistance human ovarian cancer cell line COC1/DDP and explore their correlation to the chemoresitance of COG1/DDP. 目的 探讨人卵巢癌顺铂耐药细胞株COC1/DDP中抗凋亡蛋白Bcl-XL、Bcl-2、细胞色素C的表达及半胱天冬氨酸蛋白酶-3(caspase-3)活性与人卵巢癌顺铂耐药的关系。
- ATRA can both inhibit proliferation and induce differentiation of human ovarian cancer cell line COC1.These results may provide an effective and novel approach for clinical treatment of human ovarian cancer. ATRA能明显抑制人卵巢癌细胞株COC1的生长增殖并诱导部分细胞向良性分化。
