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Corticosteroids also increase the deposition of ECM in the trabecular meshwork leading to decreased outflow facility. 类固醇也能够通过增加细胞外基质的沉积导致房水流畅系数的下降。
The effect was thought to be mediated via trabecular meshwork glucocorticoid receptors. 这种效应被认为是通过小梁网的糖皮质激素受体介导的。
The search led to the identification of mutations in a gene that encodes for a trabecular meshwork protein (TIGR). 这项研究，鉴定出编码小梁网蛋白（TIGR）的突变基因。
Trabecular meshwork,ciliary body,retina and optic nerve will be the targets for glaucoma gene therapy. 青光眼基因治疗的靶目标包括眼前节的小梁网和睫状体、眼后节的视网膜神经节细胞等。
The mechanism of elevated intraocular pressure is increased aqueous outflow resistance owing to an accumulation of extracellular matrix material in the trabecular meshwork. 眼压升高的机制被认为是由于小梁网细胞外基质的沉积引起的房水流出阻力增加。
Changes in the microstructure of the trabecular meshwork may cause a decrease in outflow facility and an increase in IOP. 小梁网显微结构的改变能够引起房水流畅系数的降低而升高眼压。
Glucocorticoid effects on the human trabecular meshwork can be used as a model system in which to study glaucomatous damage to the trabecular meshwork. 糖皮质激素对小梁网的作用可作为一个良好模型用于研究青光眼的发病机制。
The apoptosis of retinal ganglion cells and trabecular meshwork cells plays an important role in the pathogenesis,diagnosis and management of glaucoma. 青光眼视网膜神经节细胞及小梁细胞的凋亡在青光眼的发病机制、诊断治疗中发挥重要作用。
Trabecular meshwork (TM) transplantation is a replacement of the diseased TM by the functioning TM to lower intraocular pressure. 小梁移植是移植有功能的小梁取代病损小梁，以达到降眼压的目的。
The role that protein expression regulation of trabecular meshwork cells and the related signal transduction pathways play in the pathogenesis of glaucoma. 小梁细胞蛋白表达调控和相关信号传导通路在青光眼发病过程中的作用。
Levels of tissue plasminogen activator, stromelysin [34], and metalloproteases [38,39] have been shown to decrease in trabecular meshwork cultures treated with dexamethasone. 在地塞米松处理过的小梁细胞中组织纤维蛋白溶酶原激活剂、基质降解酶[34]和金属蛋白酶[38,39]水平下降。
Furthermore, dexamethasone treatment inhibits trabecular meshwork cell arachadonic acid metabolism [40] and reduces the phagocytic properties of the cells [38,41]. 而且，地塞米松可以抑制小梁细胞花生四烯酸的代谢[40]，从而降低细胞的吞噬功能[38,41]。
The acid phosphatase alkaline phosphatase lactate dehydrogenase and succinate dehydrogenase in the endothelium on the trabecular meshwork in primary glaucoma were studied. 取原发性青光眼小梁网组织、察其中酸性磷酸酶、性磷酸酶、酸脱氢酶、珀酸脱氢酶的活性。
Overexpression of certain gene(s)in trabecular meshwork to increase the resistance to aqueous humor outflow would be an ideal method to create an animal model of glaucoma. 将目的基因导入小梁组织使房水排出阻力增加是一种理想的造模方法。
In our experiment, genistein lead to a decrease in [Ca2+]i of TMC, so inferring that it can relax the trabecular meshwork ,which can lead to the increase of outflow facility. 本试验中genistein作用于小梁细胞后引起[Ca2+]i呈剂量依赖性降低，由此认为genistein通过降低小梁细胞[Ca2+]i，从而松弛小梁网，促进房水流出。
The authors treated the trabecular meshwork of 8 monkey eyes with the argon laser by a variety of energy in an attempt to cause moderate, consistent intraocular pressure (IOP) elevation. 摘要本报告为首次以猕猴作雷射诱发青光眼之实验，以探讨最理想之各项雷射参数及能量，以期达到最佳之效果。
Conclusion The expression of AQP-1 is inhibited by Dex in bovine trabecular meshwork cells. It may be one of reasons that retard the aqueous outflow in corticosteroid induced glaucoma. 结论地塞米松使培养的牛眼小梁细胞AQP-1的表达减少,这可能是皮质类固醇性青光眼房水流出阻力增加的原因之一。
Purpose: To determine the effects of adenovirus -delivered exoenzyme C3 transferase (C3) gene expression in normal or dexamethasone (DEX)-treated human trabecular meshwork (HTM) cell culture. 目的:探讨腺病毒载体介导的外酶C3转移酶(exoenzyme C3transferase,C3)表达对体外培养的正常或地塞米松处理的人类小梁细胞的作用。
The titanium-sapphire laser also causes less thermal damage in the trabecular meshwork than argon laser trabeculoplasty (ALT) or selectie laser trabeculoplasty (SLT), the studies showed. 研究显示，与氩激光小梁成形术（ALT）或选择性激光小梁成形术（SLT）相比，钛宝石激光小梁成形术引起小梁网热损伤较小。
The corticosteroid dexamethasone increases glycosaminoglycan, elastin, and fibronectin production in cultured trabecular meshwork; the glycosaminoglycan deposition increases further with prolonged steroid exposure [33,34]. 地塞米松可以引起培养的小梁细胞分泌葡萄糖胺聚糖、弹力蛋白和纤维连接蛋白的增加；葡萄糖胺聚糖的沉积会随着地塞米松作用时间的延长而增加[33,34]。