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- 肝癌细胞HepG2HepG2 cell
- 人肝癌细胞HepG2Human hepatocellular carcinoma HepG2
- PNP/fludarabine自杀基因系统对人肝癌细胞HepG2体外杀伤效应The Killing Effect of PNP/fludarabine Suicide Gene System on Human Hepatocellular-carcinoma Cell HepG2 in vitro
- 细胞cell
- 目的观察多氯联苯(PCBs)153和苯并[a]芘(B[a]P)单独及联合处理对人肝肿瘤细胞HepG2的DNA损伤作用。Objective To explore effects of polychlorinated biphenyl, PCB153 on DNA damage induced by benzo(a)pyrene (B[a]P) in HepG2 cells.
- 人淋巴因子激活的杀伤细胞对肝癌细胞株凋谢作用的观察A Observaion of Apoptoisi of Liver Cancer Cells Induced by Human LAK Cells
- 单独检测24只种植耐药细胞HepG2/ADM的裸鼠在服用溴隐亭前后分别注射的99mTc-MIBI在肝脏肿瘤部位的潴留情况。99mTc-MIBI SPECT was performed to detect the tumor 99mTc-MIBI accumulation index before and after the BCT treatment.
- 利用噬菌体表面展示技术构建人肝癌细胞的cDNA表达文库Construction of a phage-displayed library with cDNA of human hepatocarcinoma cell
- 肝癌细胞HCC; hepatoma carcinoma cell
- 东湖、汉江、长江是武汉市主要水源 ,本文研究了该三种水源的氯化饮水有机提取物对人肝肿瘤细胞HepG2的脂质过氧化作用 (LPO) ,探讨氯化饮水有机提取物引起细胞损伤的机制。Dong(D)lake,Han(H)river and Yangtze(Y)river are main water supplies of the city of Wuhan. In the present study the effect of organic extracts of chlorinated drinking water(CDW)processed from raw water of D lake,H river and Y river on lipid peroxidation(LPO)was evaluated in human HepG2 cells to explore the possible mechanism of cell damage caused by CDW extracts.
- 肝刺激因子对人肝癌细胞BEL -7402p21~(ras)表达的影响Influence of hepatic stimulator substance on p21~(ras) expression in human hepatic carcinoma cells BEL-7402
- 人肝癌细胞human hepatocarcinoma cell
- 肝癌细胞H22H22
- H22肝癌细胞Liver cancer H22 cell
- 目的探讨携带多药耐药基因1(multidrug resistance gene 1,MDR1)反义RNA重组腺病毒载体在裸鼠体内逆转肝癌多药耐药细胞HepG2/阿霉素(adriamycin,ADM)的疗效及作用机制。Objective To investigate if an adenovirus vector carrying antisence multidrug resistance gene 1 (MDR1) could reverse multidrug resistance (MDR) of HepG2/ adriamycin (ADM) cells in tumors transplanted in athymic mice. Methods An adenovirus vector carrying AFP promoter and antisence MDR1 was constructed.
- 如肝癌细胞H22 cells
- 鼠肝癌细胞H4IIE cell line
- 肝癌细胞株Hep G2
- 肝癌细胞RNAHepatic cancer cell RNA
- 抗肝癌细胞synergistic inhibition
